[1]
|
Spiegel S, Foster D, Kolesnick R. Signal transduction through lipid second messengers[J]. Curr Opin Cell Biol,1996, 8(2):159~167. |
[2]
|
Merrill AH Jr, Schmelz EM,Dillehay DL, et al. Sphingolipids- the enigmatic lipid class:biochemistry, physiology, and pathophysilogy[J]. Toxicol Appl Pharmacol,1997, 142(1):208~225. |
[3]
|
Tepper AD,Cock JG,de Vries E,et al.CD 95/Fas-induced ceramide formation proceeds with slow kinetics and is not blocked by caspase-3/CPP32 inhibition[J]. J Biol Chem,1997, 272(39):24308~24312. |
[4]
|
Boland MP, Foster SJ, O'Neill LA. Daunorubicin activates N FkappaB and induces kappaB-dependent gene expression in HL-60 promyelocytic and Jurkat T lymphoma cell[J]. J Biol Chem,1997, 272(20):12952~12960. |
[5]
|
Verheij M,Bose R, Lin XH, et al. Requirement for ceramide-initiated SAPK/JNK signaling in stress-induced apoptosis[J]. Nature, 1996, 380(6569):75~79. |
[6]
|
Michael JM, Lavin MF, Watters DJ. Resistance to radiation-induced apoptosis in Burkitt's lymphoma cell is associated with defective ceramide signaling[J]. Canaer Res,1997, 57(16):3600~3605. |
[7]
|
Kimura K,Bowen C,Spiegal S,et al.Tumor necrosis factor-α sensitizes prostate cancer cells to γ-irradiation-induced apoptosis[J]. Canaer Res, 1999, 59:1606~1614. |
[8]
|
Vidair CA Chen CH,ing CC,et al.Apoptosis induced by X-irradiation of rec-myc cell is postmitotic not predicted by the time after irradiation or behavior of sister cells[J]. Cancer Res, 1996, 56:4116~4118. |
[9]
|
Chmura SJ,Mauceri HJ,Advani S,et al.Decreasing the apoptotic threshold of tumor cells through protein kinase C inhibition and sphingomyelinase activation incrases tumor killing by ionizing radiation[J]. Cancer Res, 1997, 57(19):4340~4347. |
[10]
|
Bruno AP,Laurent G,Averbeck D,et al.Lack of ceramide generation in TF-1 human myeloid leukemic cells resistant to ionizing radiation[J]. Cell Death Differ, 1998, 5(2):172~182. |
[11]
|
Chmura SJ, Nodzenski E, Weichselbaum RR, et al. Protrin kinase C inhibition induces apoptosis and ceramide production through activation of a neutral sphingomyelinase[J]. Cancer Res, 1996, 56(12):2711~2714. |
[12]
|
Chmura SJ,Nodzenski E,Crane MA,et al.Cross-talk between ceramide and PKC activity in the control of apoptosis in W EHI-231[J]. Adv Exp Med Biol, 1996, 406:39~55. |
[13]
|
Haimovtz-Friedman A. Radiation-induced signal transduction and stress response[J]. Radiat Res,1998, 150(5 Suppl):S102~S108. |
[14]
|
Nyberg L, Duan RD, Axelson J, et al. Identification of an alkaline sphingomyelnase activity in human bile[J]. Biochim Biophys Acta, 1996, 1300(1):42~48. |
[15]
|
Chmura SJ,Nodzenski E,Beckett MA et al.Loss of ceramide production confers resistance to radiaion-induced apoptosis[J]. Cancer Res, 1997, 57(7):1270~1275. |
[16]
|
Santana P,Pena LA,Haimovitz-Friedman A,et al.Acid sphingomyelinase-deficient human lympoblasts and mice are defective in radiation-induced apoptosis[J]. 1996, 86(2):189~199. |
[17]
|
Burrow M E, Weldon CB, Tangy, et al. Differences in susceptibility to tumor necrosis factr-α-induced apoptosis among MCF-7 breast cancer cell variants[J]. Cancer Res, 1998, 58:4940~4946. |
[18]
|
Cai Z, Bettaieb A, Mahdani N ET, et al. Alteration of the sphingomyelin/ceramide pathway is associated with resistance of human breast carcinoma MCF7 cells to tumor necrosis factor-α-mediated cytotoxicity[J]. J Biol Chem, 1997, 272:6918~6926. |