[1] Alvarez-Gonzalez R,Pacheco-Rodriguez G,Mendoza-Alvarez H.Enzymology of ADP-ribose polymer synthesis.Mol Cell Biochem,1994,138(1-2):33-37.
[2] El-Khamisy SF,Masutani M,Suzuki H,et al.A requirement for PARP-1 for the assembly or stability of XRCC1 nuclear foci at sites of oxidative DNA damage.Nucleic Acids Res.2003,31(19):5526-5533.
[3] Okano S,Kanno SI,Nakajima S,et al.Cellular responses and repair of single-strand breaks introduced by UV damage endonuclease in mammalian cell.J Biol Chem,2000,275(42):32635-32641.
[4] Leppard J,Dong Z,Marckey ZB,et al.Physical and functional interaction between DNA ligase Ⅲalpha and poly (ADP-ribose) polymerase 1 in DNA single-strand break repair.Mol Cell Biol,2003,23(16):5919-5927.
[5] Wesierska-Gadek J,Wang ZQ,Schmid G,et al.Reduced stability of regularly spliced but not alternatively spliced p53 protein in PARP-deficient mouse fibroblasts.Cancer Res,1999,59(1):28-34.
[6] Fujita T,Maruyama M,Araya J,et al.Hydrogen peroxide induces upregulation of Fas in human airway epithelial cells via the activation of PARP-p53 pathway.Am J Repair Cell Mol Biol,2002,27(5):542-552.
[7] Wesierska-Gadek J,Wojcieehowski J,Schmid G.Central and carboxy-terminal regions of human p53 protein are essential for interaction and complex formation with PARP-1.J Cell Biochem,2003,89(2):220-232.
[8] SüsseS,ScholzCJ,BürkleA,etal.Poly (ADP-ribose)polymerase(PARP-1) and p53 independently function in regulating doublestrand break repair in primate cells.Nucl Acid Res,2004,32(2):669-680.
[9] De Blasioa A,Messinab C,Santullib A,el al.Differentiative pathway activated by 3-aminobenzamide,an inhibitor of PARP in human osteosarcoma MG-63 cells.FEBS Letter,2005,579(3):615-620.
[10] Wang ZQ,Stingl L,Morrison C,el al.PARP is important for genomic stability but dispensable in apoptosis.Genes Dev,1997,11(18):2347-2358.
[11] d'Adda di Fagagna F,Hande MP,Tong WM,et al.Functions of poly(ADP-ribose) polymerase in controlling telomere length and chromosomal stability.Nat Genet,1999,23(1):76-80.
[12] Decker P,Isenberg D,Muller S.Inhibition of caspase-3-mediated poly (ADP-ribose) polymerase (PARP) apoptotic cleavage by human PARP autoantibodies and effect on cells undergoing apoptosis.J Biol Chem,2000,275(12):9043-9046.
[13] Los M,Mozoluk M,Ferriari D,et al.Activation and caspase-mediated inhibition of PARP a molecular switch between fibroblast necrosis and apoptosis in death receptor signaling.Mol Biol Cell,2002,13(3):978-988.
[14] Germain M,Affar EB,D'Amours D,et al.Cleavage of automodified poly (ADP-ribose) polymerase during apoptosis:Evidence for involvement of caspase-7.J Biol Chem,1999,274(40):28379-28384.
[15] Alano CC,Ying W,Swanson RA.Poly (ADP-ribose) polymerase-1-mediated cell death in astrocytes requires NAD^+ depletion and mitoehondrial permeability transition.J Cell Biochem,2004,279(18):18895-18902.
[16] Veuger SJ,Curtin NJ,Richardson CJ.Radiosensitization and DNA repair inhibition by the combined use of novel inhibitors of DNA-dependent protein kinase and poly (ADP-Ribose) polymerase-1.Cancer Res,2003,63(10):6008-6015.
[17] Chalmers A,Johnston P,Woodcock M.PARP-1,PARP-2,andthe cellular response to low doses of ionizing radiation.Int J Radiat Oncol Biol Phys,2004,58(2):410-419.