[1] Kennelly JC, Lane MP, Barker JA, et al.Genotoxic activity of 1-chloromethyl-pyrenein stomach epithelium in vivo:insensitivity of the stomach scintillation UDS assay[J].Cancinogenesis, 1993, 14:637-640.
[2] Lankinen MH, Vilpo LM, Vilpo JA.UV-and gamma-irradiation-induced DN A single-strand breaks and their repair in human blood granulocytes and lymphocytes[J].Mutat Res, 1996, 352:31-38.
[3] Alapetite C, Wachter T, Sage E, et al.Use of the alkaline comet assay to detect DNA repair deficiencies in human fibroblasts exposed to UVC, UVB, UVA andgamma-rays[J].Int J Radiat Biol, 1996, 69:359-369.
[4] Wojewodzka M, Kruszewski M, Szumiel I.Anti-CD38 prevents the decelopment of the adaptive response induced by X-rays in human lymphocytes[J].Mutagenesis, 1996, 11:593-596.
[5] Wojcik A, Sauer C, Zolzer F, et al.Analysis of DNA damage recovery processes in the adaptive response to ionizing radiation in human lymphocytes[J].Mutagenesis.1996, 11:291-297.
[6] Hughes CM, Lewis SE, McKelvey Martin VJ, et al.The effects of antioxidant supplementation during percoll preparation on human sperm DN A integrity[J].Hum Reprod, 1988, 13:1240-1247.
[7] Zheng H, Olive PL.Reduction of tumor hypoxia and inhibition of DN A repair by nicotinamide after irradiation of SCCV Ⅱ murine tumors and normal tissues[J].Cancer Res, 1996, 56:2801-2808.
[8] Kent CR, Eady JJ, Ross GM, et al.The comet moment as a measure of DNA damage in the comet assay[J].Int J Radiat Biol, 1995, 67:655-660.
[9] Plappert U, Raddatz K, Roth S, et al.DN A-damage detection in man after radiation exposure-the cometasay-its possible application for human biomonitoring[J].Stem Cells Dayt, 1995, 13:215-222.
[10] Kruszewski M, Wojewodzka M, Iwanenko T, et al.Application of the comet assay for monitoring DNA damage in workers exposed to chronic low-dose irradiation.Ⅱ.Base damage[J].Mutat Res, 1998, 416:37-57.
[11] Wojewodzka M, Kruszewski M, Iwanenko T, et al.Application of the comet assay for monitoring DNA damage in workers exposed to chronic low-dose irradiation.I.Strand breakage[J].Mutat Res, 1998,416:21-35.
[12] Muller W U, Bauch T, Streffer C, et al.Comet assay studies of radiation-induced DNA damage and repair in various tumour cell lines[J].Int J Radiat Biol, 1994,65:315-319.
[13] Olive PL, Banath JP, MacPhail HS, et al.Lack of a correlation between radiosensitivity and DNA double-strand break induction or rejoining in six human tumor cell lines[J].Cancer Res, 1994, 54:3939-3946.
[14] Olive PL, Horsman MR, Grau C, et al.Detection of hypoxic cells in a C3H mouse mammary carcinoma using the comet assay[J].Br J Cancer, 1997, 76:694-699.
[15] Olive PL.Detection of hypoxia by measurement ofDNA damage in individual cells from spheroids and murine tumours exposed to bioreductive drugs.I.Tirapazamine[J].Br J Cancer, 1995, 71:529-536.
[16] Fairbairn DW, Olive PL, O'Neill KL.The comet assay:a comprehensive review[J].Mutat Res, 1995,339:37-59.
[17] Humar B, Muller H, Scott RJ.Elevated frequency of p53-independent apoptosis irradiation increases levels of DNA breaks in ataxia telangiectasia lymphoblasts[J].Int J Radiat Biol, 1997, 72:257-269.
[18] Hu Q, Hill RP.Radiosensitivity, apoptosis and repair of DNA double-strand breaks in radiation-sensitiveChinese hamster ovary cell mutants treated at different does rates[J].Radiat Res, 1996, 146:636-645.