[1]

郭宝帅, 赵雪琦, 李晓阳. 碘131 对甲状腺功能亢进的治疗进展[J/OL]. 世界最新医学信息文摘, 2018, 18(43): 83−84[2018-07-26]. http://www.doc88.com/p-1126632858297.html. DOI: 10.19613/j.cnki.1671−3141.2018.43.031.

Guo BS, Zhao XQ, Li XY. Progress in the treatment of hyperthyroidism with iodine 131[J/OL]. World Latest Medicine Information (ElectronicVersion), 2018, 18(43): 83−84[2018-07-26]. http://www.doc88.com/p-1126632858297.html. DOI: 10.19613/j.cnki.1671−3141.2018.43.031.

[2] 潘红方, 邱夏芹.  小剂量甲状腺素治疗老年甲状腺功能减退症的临床疗效[J]. 包头医学院学报, 2016, 32(8): 80-81.   doi: 10.16833/j.cnki.jbmc.2016.08.046
Pan HF, Qiu XQ.  Clinical efficacy of low-dose thyroxine in the treatment of senile hypothyroidism[J]. J Baotou Med Coll, 2016, 32(8): 80-81.   doi: 10.16833/j.cnki.jbmc.2016.08.046
[3] Alexander EK, Larsen PR.  High dose of 131I therapy for the treatment of hyperthyroidism caused by Graves' disease[J]. J Clin Endocrinol Metab, 2002, 87(3): 1073-1077.   doi: 10.1210/jcem.87.3.8333
[4] Gómez N, Gómez JM, Orti A, et al.  Transient hypothyroidism after iodine-131 therapy for Grave's disease[J]. J Nucl Med, 1995, 36(9): 1539-1542.
[5] Aizawa Y, Yoshida K, Kaise N, et al.  The development of transient hypothyroidism after iodine-131 treatment in hyperthyroid patients with Graves' disease: prevalence, mechanism and prognosis[J]. Clin Endocrinol(Oxf), 1997, 46(1): 1-5.   doi: 10.1046/j.1365-2265.1997.d01-1737.x
[6] Ashizawa K, Imaizumi M, Usa T, et al.  Metabolic cardiovascular disease risk Factors and their clustering in subclinical hypothyroidism[J]. Clin Endocrinol(Oxf), 2010, 72(5): 689-695.   doi: 10.1111/j.1365-2265.2009.03697.x
[7] Mc Quade C, Skugor M, Brennan DM, et al.  Hypothyroidism and moderate subclinical hypothyroidism are associated with increased all-cause mortality independent of coronary heart disease risk factors: a PreCIS database study[J]. Thyroid, 2011, 21(8): 837-843.   doi: 10.1089/thy.2010.0298
[8] Dias RG, Negrão CE, Krieger MH.  Nitric oxide and the cardiovascular system: cell activation, vascular reactivity and genetic variant[J]. Arq Bras Cardiol, 2011, 96(1): 68-75.
[9] Furchgott RF, Zawadzki JV.  The obligatory role of endothelial cells in the relaxation of arterial smooth muscle by acetylcholine[J]. Nature, 1980, 288(5789): 373-376.   doi: 10.1038/288373a0
[10] Ignarro LJ, Buga GM, Wood KS, et al.  Endothelium-derived relaxing factor produced and released from artery and vein is nitric oxide[J]. Proc Natl Acad Sci USA, 1987, 84(24): 9265-9269.   doi: 10.1073/pnas.84.24.9265
[11] Dias RG, Negrão CE, Krieger MH.  Nitric oxide and the cardiovascular system: cell activation, vascular reactivity and genetic variant[J]. Arq Bras Cardiol, 2011, 96(1): 68-75.
[12]

张陆燕. 促甲状腺激素(TSH)对血管内皮细胞影响的作用及机制的研究[D]. 兰州: 兰州大学, 2013.

Zhang LY. Effect and mechanism of thyroid stimulating hormone (TSH) on vascular endothelial cells[D]. Lanzhou: Lanzhou University, 2013.

[13] 陈刚, 廖志良, 刘笃秋, 等.  亚临床甲状腺功能减退症与冠状动脉粥样硬化性心脏病的相关性[J]. 海南医学, 2017, 28(3): 372-373.   doi: 10.3969/j.issn.1003-6350.2017.03.009
Chen G, Liao ZL, Liu DQ, et al.  Relationship between subclinical hypothyroidism and coronary atherosclerotic heart disease[J]. Hainan Med J, 2017, 28(3): 372-373.   doi: 10.3969/j.issn.1003-6350.2017.03.009
[14] Hosseini M, Dastghaib SS, Rafatpanah H, et al.  Nitric oxide contributes to learning and memory deficits observed in hypothyroid rats during neonatal and juvenile growth[J]. Clinics (Sao Paulo), 2010, 65(11): 1175-1181.   doi: 10.1590/S1807-59322010001100021
[15] McAllister RM, Albarracin I, Price EM, et al.  Thyroid status and nitric oxide in rat arterial vessels[J]. J Endocrinol, 2005, 185(1): 111-119.   doi: 10.1677/joe.1.06022
[16] Quesada A, Sainz J, Wangensteen R, et al.  Nitric oxide synthase activity in hyperthyroid and hypothyroid rats[J]. Eur J Endocrinol, 2002, 147(1): 117-122.   doi: 10.1530/eje.0.1470117
[17] Engin AB, Sepici-Dincel A, Gonul II, et al.  Oxidative stress-induced endothelial cell damage in thyroidectomized rat[J]. Exp Toxicol Pathol, 2012, 64(5): 481-485.   doi: 10.1016/j.etp.2010.11.002
[18] Torlak V, Zemunik T, Modun D, et al.  131I-induced changes in rat thyroid gland function[J]. Braz J Med Biol Res, 2007, 40(8): 1087-1094.   doi: 10.1590/S0100-879X2006005000127
[19]

韦凌云, 杨丕坚, 李舒敏, 等. 亚临床甲状腺功能减退症患者血管内皮功能与促甲状腺激素的相关性[J]. 中华实用诊断与治疗杂志, 2014, 28(2): 191-192. DOI: 10.13507/j.issn.1674-3474.2014.02.037.

Wei LY, Yang PJ, Li SM, et al. Correlation between vascular endothelial function and thyroid stimulating hormone in patients with subclinical hypothyroidism[J]. J Chin Pract Diagn Ther, 2014, 28(2): 191-192. DOI: 10.13507/j.issn.1674-3474.2014.02.037.

[20]

Gao C, Li T, Liu J, et al. Endothelial Functioning and Hemodynamic Parameters in Rats with Subclinical Hypothyroid and the Effects of Thyroxine Replacement[J/OL]. PLoS One, 2015, 10(7): e0131776[2018-07-26]. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4497722. DOI: 10.1371/journal.pone.0131776.

[21] Sarati LI, Martinez CR, Artés N, et al.  Hypothyroidism: age-related influence on cardiovascular nitric oxide system in rats[J]. Metabolism, 2012, 61(9): 1301-1311.   doi: 10.1016/j.metabol.2012.01.022
[22] Fellet AL, Arza P, Arreche N, et al.  Nitric oxide and thyroid gland: modulation of cardiovascular function in autonomic-blocked anaesthetized rats[J]. Exp Physiol, 2004, 89(3): 303-312.   doi: 10.1113/expphysiol.2004.027201
[23] Lacoste-Collin L, Jozan S, Pereda V, et al.  Influence of A Continuous Very Low Dose of Gamma-Rays on Cell Proliferation, Apoptosis and Oxidative Stress[J]. Dose Response, 2015, 13(1): 1-17.   doi: 10.2203/dose-response.14-010.Lacoste-Collin
[24]

Ibuki Y, Goto R. Ionizing radiation-induced macrophage activation: augmentation of nitric oxide production and its significance[J/OL]. Cell Mol Biol (Noisy-le-grand), 2004: 617-626[2018-07-26]. https://www.ncbi.nlm.nih.gov/pubmed/?term=15579255.

[25] Nowosielska EM, Wrembel-Wargocka J, Cheda A, et al.  Enhanced cytotoxic activity of macrophages and suppressed tumor metastases in mice irradiated with low doses of X-rays[J]. J Radiat Res, 2006, 47(3/4): 229-236.
[26] Rodríguez-Gómez I, Moliz JN, Quesada A, et al.  L-Arginine metabolism in cardiovascular and renal tissue from hyper-and hypothyroid rats[J]. Exp Biol Med (Maywood), 2016, 241(5): 550-556.   doi: 10.1177/1535370215619042