[1] Straub JM, New J, Hamilton CD, et al. Radiation-induced fibrosis:mechanisms and implications for therapy[J]. J Cancer Res Clin Oncol, 2015, 141(11):1985-1994. DOI:10.1007/s00432-015-1974-6.
[2] 郑啓盛, 刘培勋.放射性肺纤维化的分子机制及其防治药物综述[J].辐射研究与辐射工艺学报, 2016, 34(1):1-10. DOI:10.11889/j.1000-3436.2016.rrj.34.010101.
Zheng QS, Liu PX. Review on molecular mechanisms and protective drugs research on radiation-induced lung fibrosis[J]. J Radiat Res Radiat Process, 2016, 34(1):1-10.  doi: 10.11889/j.1000-3436.2016.rrj.34.010101
[3] 朱玉凤.肺纤维化细胞因子机制研究进展[J].当代医药论丛, 2015, 13(5):157-157, 158.
Zhu YF. Progress of cytokine mechanism in pulmonary fibrosis[J]. Contemporary Med Forum, 2015, 13(5):157-157, 158.
[4] 王司仪, 原铭贞, 刘笑玎, 等.细胞因子与肺纤维化相关性的研究进展[J].吉林大学学报(医学版), 2014, 40(6):1325-1329. DOI:10.13481/j.1671-587x.20140641.
Wang SY, Yuan MZ, Liu XD, et al. Advance research on relationship between cytokines and pulmonary fibrosis[J]. J Jilin Univ Med Edit, 2014, 40(6):1325-1329.  doi: 10.13481/j.1671-587x.20140641
[5] Leask A, Abraham DJ. TGF-beta signaling and the fibrotic response[J]. FASEB J, 2004, 18(7):816-827. DOI:10.1096/fj.03-1273rev.
[6] Hall BE, Wankhade UD, Konkel JE, et al. Transforming growth factor-β3(TGF-β3) knock-in ameliorates inflammation due to TGF-β1 deficiency while promoting glucose tolerance[J]. J Biol Chem, 2013, 288(44):32074-32092. DOI:10.1074/jbc.M113. 480764.
[7] Ask K, Bonniaud P, Maass K, et al. Progressive pulmonary fibrosis is mediated by TGF-β isoform 1 but not TGF-β3[J]. Int J Biochem Cell Biol, 2008, 40(3):484-495. DOI:10.1016/j.biocel.2007. 08. 016.
[8]

Xu L, Xiong S, Guo R, et al. Transforming growth factor β3 attenuates the development of radiation-induced pulmonary fibrosis in mice by decreasing fibrocyte recruitment and regulating IFN-γ/IL-4 balance[J]. Immunol Lett, 2014, 162(1 Pt A):27-33.DOI:10.1016/j.imlet.2014.06.010.
[9] Horowitz JC, Thannickal VJ. Epithelial-mesenchymal interactions in pulmonary fibrosis[J]. Semin Respir Crit Care Med, 2006, 27(6):600-612. DOI:10.1055/s-2006-957332.
[10] 杜雪梅, 柳晓兰, 崔玉芳, 等.放射性肺损伤小鼠动物模型的建立及其病变规律[J].中国体视学与图像分析, 2003, 8(4):203-206. DOI:10.3969/j.issn.1007-1482.2003.04.003.
Du XM, Liu XL, Cui YF, et al. ESTABLISHMENT OF IRRADIA-TION-INDUCED LUNG INJURY MOUSE MODEL AND ITS PATHOLOGICAL CHANGES REGULARITY[J]. CHIN J STERE-OL IMAGE ANALYSIS, 2003, 8(4):203-206.  doi: 10.3969/j.issn.1007-1482.2003.04.003
[11] 杨燕光, 王金云, 张珏, 等.局部晚期非小细胞肺癌调强放疗与三维适形放疗的剂量学及临床疗效分析[J].中国肿瘤, 2014, 23(10):873-877. DOI:10.11735/j.issn.1004-0242.2014.10.A017.
Yang YG, Wang JY, Zhang J, et al. An Analysis of Dosimetric and Clinical Effect of Intensity Modulated Radiotherapy and Three-dimensional Conformal Radiotherapy for Locally Advanced Non-small Cell Lung Cancer[J]. China Cancer, 2014, 23(10):873-877.  doi: 10.11735/j.issn.1004-0242.2014.10.A017
[12] Xiong SS, Guo RF, Yang ZH, et al. Regulatory T Cells Promote β-Catenin-Mediated Epithelium-to-Mesenchyme Transition During Radiation-Induced Pulmonary Fibrosis[J]. Int J Radiat Oncol Biol Phys, 2015, 93(2):425-435. DOI:10.1016/j.ijrobp.2015.05.043.
[13] 陈忠杰, 王莉, 方银善, 等.放射线剂量和剂量率对小鼠肺纤维化的影响[J].中华结核和呼吸杂志, 2016, 39(2):117-121. DOI:10.3760/cma.j.issn.1001-0939.2016.02.010.
Chen ZJ, Wang L, Fang YS, et al. Effect of radiation dose and dose rate on pulmonary fibrosis in mice[J]. Chin J Tubere Respir Dis, 2016, 39(2):117-121.  doi: 10.3760/cma.j.issn.1001-0939.2016.02.010
[14]

赵利强. 沙百润肺颗粒干预大鼠放射性肺纤维化的实验研究[D]. 太原: 山西省中医药研究院, 2016: 1-46.

Zhao LQ. The experimental study of Shabai Moistening Lung particles intervening in radiation-induced pulmonary fibrosis in rats[D]. Taiyuan: Shanxi Traditional Chinese Medicine Institute, 2016: 1-46.
[15] Reilkoff RA, Bucala R, Herzog EL. Fibrocytes:emerging effector cells in chronic inflammation[J]. Nat Rev Immunol, 2011, 11(6):427-435. DOI:10.1038/nri2990.
[16]

熊珊珊. 调节性T淋巴细胞在放射性肺纤维化中的作用及机制研究[D]. 北京: 军事医学科学院, 2015: 1-135.

Xiong SS. The roles and mechanisms of Regulatory T cells (Tregs) in radiation-induced pulmonary fibrosis[D]. Beijing: Academy of Military Medical Sciences, 2015: 1-135.
[17] Hay ED. The mesenchymal cell, its role in the embryo, and the remarkable signaling mechanisms that create it[J]. Dev Dyn, 2005, 233(3):706-720. DOI:10.1002/dvdy.20345.
[18]

徐龙. 转化生长因子-β3调控循环纤维细胞在放射性肺纤维化中的作用研究[D]. 北京: 军事医学科学院, 2015: 1-101.

Xu L. Study on the role of transforming growth factor-β3 by regulating fibrocytes in radiation-induced pulmonary fibrosis[D]. Beijing: Academy of Military Medical Sciences, 2015: 1-101.